Pharmacological inhibition of aberrant transcription factor complexes in inversion 16 acute myeloid leukemia

Sandeep Potluri; Daniel Coleman; Constanze Bonifer

doi:10.21037/sci.2018.09.03

Pharmacological inhibition of aberrant transcription factor complexes in inversion 16 acute myeloid leukemia

Sandeep Potluri, Daniel Coleman, Constanze Bonifer

Research output: Contribution to journal › Editorial

1 Citation (Scopus)

Abstract

Within the hematopoietic system, core binding factor beta (CBFβ) normally forms a complex with the master hematopoietic regulator, RUNX1 (1) and stabilizes its binding to DNA (2).

Original language	English
Article number	30
Number of pages	5
Journal	Stem Cell Investigation
Volume	5
DOIs	https://doi.org/10.21037/sci.2018.09.03
Publication status	Published - 30 Sept 2018

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title = "Pharmacological inhibition of aberrant transcription factor complexes in inversion 16 acute myeloid leukemia",

abstract = "Within the hematopoietic system, core binding factor beta (CBFβ) normally forms a complex with the master hematopoietic regulator, RUNX1 (1) and stabilizes its binding to DNA (2).",

author = "Sandeep Potluri and Daniel Coleman and Constanze Bonifer",

year = "2018",

month = sep,

day = "30",

doi = "10.21037/sci.2018.09.03",

language = "English",

volume = "5",

journal = "Stem Cell Investigation",

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AU - Potluri, Sandeep

AU - Coleman, Daniel

AU - Bonifer, Constanze

PY - 2018/9/30

Y1 - 2018/9/30

N2 - Within the hematopoietic system, core binding factor beta (CBFβ) normally forms a complex with the master hematopoietic regulator, RUNX1 (1) and stabilizes its binding to DNA (2).

AB - Within the hematopoietic system, core binding factor beta (CBFβ) normally forms a complex with the master hematopoietic regulator, RUNX1 (1) and stabilizes its binding to DNA (2).

U2 - 10.21037/sci.2018.09.03

DO - 10.21037/sci.2018.09.03

M3 - Editorial

C2 - 30363728

SN - 2306-9759

VL - 5

JO - Stem Cell Investigation

JF - Stem Cell Investigation

M1 - 30

ER -

Pharmacological inhibition of aberrant transcription factor complexes in inversion 16 acute myeloid leukemia

Abstract

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