Shifting hemodynamics of blood pressure control during prolonged mental stress.

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Shifting hemodynamics of blood pressure control during prolonged mental stress. / Ring, Christopher; Burns, Victoria; Carroll, Douglas.

In: Psychophysiology, Vol. 39, No. 5, 01.09.2002, p. 585-90.

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@article{5a759094c06f463fac926d3b8a4a2f9c,
title = "Shifting hemodynamics of blood pressure control during prolonged mental stress.",
abstract = "The present study examined the hemodynamics underlying blood pressure elevations for evidence of a shift in the control of blood pressure during prolonged mental stress. Mean arterial pressure (MAP), cardiac output (CO), and total peripheral resistance (TPR) were measured at rest, during a 28-min mental arithmetic stress task, and during recovery, in 30 young healthy men and women. The stress task elicited a sustained increase in MAP: CO rose during the first half of the task but returned to baseline levels during the last quarter of the task, whereas TPR increased as the task progressed. When participants' hemodynamic reactions were classified as cardiac, vascular, or neither, there were more cardiac reactors early relative to late in the task, whereas there were more vascular reactors late relative to early. Thus, the sustained pressor response was initially supported mainly by cardiac mechanisms but subsequently by predominantly vascular mechanisms.",
author = "Christopher Ring and Victoria Burns and Douglas Carroll",
year = "2002",
month = sep,
day = "1",
doi = "10.1017.S0048577202011320",
language = "English",
volume = "39",
pages = "585--90",
journal = "Psychophysiology",
issn = "0048-5772",
publisher = "Wiley Online Library",
number = "5",

}

RIS

TY - JOUR

T1 - Shifting hemodynamics of blood pressure control during prolonged mental stress.

AU - Ring, Christopher

AU - Burns, Victoria

AU - Carroll, Douglas

PY - 2002/9/1

Y1 - 2002/9/1

N2 - The present study examined the hemodynamics underlying blood pressure elevations for evidence of a shift in the control of blood pressure during prolonged mental stress. Mean arterial pressure (MAP), cardiac output (CO), and total peripheral resistance (TPR) were measured at rest, during a 28-min mental arithmetic stress task, and during recovery, in 30 young healthy men and women. The stress task elicited a sustained increase in MAP: CO rose during the first half of the task but returned to baseline levels during the last quarter of the task, whereas TPR increased as the task progressed. When participants' hemodynamic reactions were classified as cardiac, vascular, or neither, there were more cardiac reactors early relative to late in the task, whereas there were more vascular reactors late relative to early. Thus, the sustained pressor response was initially supported mainly by cardiac mechanisms but subsequently by predominantly vascular mechanisms.

AB - The present study examined the hemodynamics underlying blood pressure elevations for evidence of a shift in the control of blood pressure during prolonged mental stress. Mean arterial pressure (MAP), cardiac output (CO), and total peripheral resistance (TPR) were measured at rest, during a 28-min mental arithmetic stress task, and during recovery, in 30 young healthy men and women. The stress task elicited a sustained increase in MAP: CO rose during the first half of the task but returned to baseline levels during the last quarter of the task, whereas TPR increased as the task progressed. When participants' hemodynamic reactions were classified as cardiac, vascular, or neither, there were more cardiac reactors early relative to late in the task, whereas there were more vascular reactors late relative to early. Thus, the sustained pressor response was initially supported mainly by cardiac mechanisms but subsequently by predominantly vascular mechanisms.

U2 - 10.1017.S0048577202011320

DO - 10.1017.S0048577202011320

M3 - Article

C2 - 12236324

VL - 39

SP - 585

EP - 590

JO - Psychophysiology

JF - Psychophysiology

SN - 0048-5772

IS - 5

ER -