Life events and haemodynamic reactions to acute mental stress in middle-aged and elderly men and women

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@article{148dfbfbdc3340df916f864b56e8c7a4,
title = "Life events and haemodynamic reactions to acute mental stress in middle-aged and elderly men and women",
abstract = "Recent versions of the reactivity hypothesis, which consider it to be the product of stress exposure and exaggerated hemodynamic reactions to stress that confers cardiovascular disease risk, assume that reactivity is independent of the experience of stressful life events. This assumption was tested in two substantial cohorts, one middle-aged and one elderly. Participants had to indicate from a list of major stressful life events up to six they had experienced in the previous 2 years. They were also asked to rate how disruptive and stressful they were, at the time of occurrence and now. Blood pressure and pulse rate were measured at rest and in response to acute mental stress. Those who rated the events as highly disruptive at the time of exposure and now exhibited blunted systolic blood pressure reactions to acute stress. The present results suggest that acute stress reactivity may not be independent of stressful life events experience.",
keywords = "reactivity, blood pressure, life events, acute stress",
author = "Douglas Carroll and Anna Phillips and Christopher Ring and G Der and K Hunt",
year = "2005",
month = may,
day = "1",
doi = "10.1111/j.1469-8986.2005.00282.x",
language = "English",
volume = "42",
pages = "269--276",
journal = "Psychophysiology",
issn = "0048-5772",
publisher = "Wiley Online Library",

}

RIS

TY - JOUR

T1 - Life events and haemodynamic reactions to acute mental stress in middle-aged and elderly men and women

AU - Carroll, Douglas

AU - Phillips, Anna

AU - Ring, Christopher

AU - Der, G

AU - Hunt, K

PY - 2005/5/1

Y1 - 2005/5/1

N2 - Recent versions of the reactivity hypothesis, which consider it to be the product of stress exposure and exaggerated hemodynamic reactions to stress that confers cardiovascular disease risk, assume that reactivity is independent of the experience of stressful life events. This assumption was tested in two substantial cohorts, one middle-aged and one elderly. Participants had to indicate from a list of major stressful life events up to six they had experienced in the previous 2 years. They were also asked to rate how disruptive and stressful they were, at the time of occurrence and now. Blood pressure and pulse rate were measured at rest and in response to acute mental stress. Those who rated the events as highly disruptive at the time of exposure and now exhibited blunted systolic blood pressure reactions to acute stress. The present results suggest that acute stress reactivity may not be independent of stressful life events experience.

AB - Recent versions of the reactivity hypothesis, which consider it to be the product of stress exposure and exaggerated hemodynamic reactions to stress that confers cardiovascular disease risk, assume that reactivity is independent of the experience of stressful life events. This assumption was tested in two substantial cohorts, one middle-aged and one elderly. Participants had to indicate from a list of major stressful life events up to six they had experienced in the previous 2 years. They were also asked to rate how disruptive and stressful they were, at the time of occurrence and now. Blood pressure and pulse rate were measured at rest and in response to acute mental stress. Those who rated the events as highly disruptive at the time of exposure and now exhibited blunted systolic blood pressure reactions to acute stress. The present results suggest that acute stress reactivity may not be independent of stressful life events experience.

KW - reactivity

KW - blood pressure

KW - life events

KW - acute stress

UR - http://www.scopus.com/inward/record.url?scp=20544456049&partnerID=8YFLogxK

U2 - 10.1111/j.1469-8986.2005.00282.x

DO - 10.1111/j.1469-8986.2005.00282.x

M3 - Article

C2 - 15943680

VL - 42

SP - 269

EP - 276

JO - Psychophysiology

JF - Psychophysiology

SN - 0048-5772

ER -