Abstract
The contribution of triacylglycerol to energy provision in the hypertrophied heart, mediated through lipoprotein lipase (LPL) is largely unknown and the contribution of very-low-density lipoprotein (VLDL) receptor to control of LPL presentation at the endothelium is unclear. For isolated perfused rat hearts, cold acclimation (CA) induced volume-overload hypertrophy, with decreased developed pressure (P
Original language | English |
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Pages (from-to) | 618-26 |
Number of pages | 9 |
Journal | Biochimica et Biophysica Acta |
Volume | 1781 |
Issue number | 10 |
DOIs | |
Publication status | Published - 1 Oct 2008 |
Keywords
- Cardiac hypertrophy
- AMPK
- Very-low-density lipoprotein receptor
- Triacylglycerol