Can genetic risk information enhance motivation for smoking cessation? An analogue study

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Can genetic risk information enhance motivation for smoking cessation? An analogue study. / Wright, AJ; French, David; Weinman, J; Marteau, TM.

In: Health Psychology, Vol. 25, 01.01.2006, p. 740-752.

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@article{91279c16a66c42c2bb11aa3a2c6373d8,
title = "Can genetic risk information enhance motivation for smoking cessation? An analogue study",
abstract = "Protection motivation theory and the extended parallel processing model are used to predict the motivational impact of information regarding a genetic susceptibility to heart disease. One hundred ninety-eight smokers read 1 of 3 vignettes: gene positive, gene negative, or standard smoking risk information. Analyses examined whether the impact of type of risk information was moderated by smokers' self-efficacy (SE) levels. Key outcomes were intention to quit and intention to attend an information session about quitting. There were significant main effects of SE and of receiving gene-positive risk information on intentions to quit. There was a significant Risk x SE interaction on intentions to attend an information session. SE was not associated with intentions to attend the information session for smokers in the gene-positive group. Intentions to attend the session were negatively associated with SE for smokers in the lower risk groups. Implications for using genetic risk information to motivate smoking cessation are discussed.",
author = "AJ Wright and David French and J Weinman and TM Marteau",
year = "2006",
month = jan,
day = "1",
doi = "10.1037/0278-6133.25.6.740",
language = "English",
volume = "25",
pages = "740--752",
journal = "Health Psychology",
issn = "0278-6133",
publisher = "American Psychological Association",

}

RIS

TY - JOUR

T1 - Can genetic risk information enhance motivation for smoking cessation? An analogue study

AU - Wright, AJ

AU - French, David

AU - Weinman, J

AU - Marteau, TM

PY - 2006/1/1

Y1 - 2006/1/1

N2 - Protection motivation theory and the extended parallel processing model are used to predict the motivational impact of information regarding a genetic susceptibility to heart disease. One hundred ninety-eight smokers read 1 of 3 vignettes: gene positive, gene negative, or standard smoking risk information. Analyses examined whether the impact of type of risk information was moderated by smokers' self-efficacy (SE) levels. Key outcomes were intention to quit and intention to attend an information session about quitting. There were significant main effects of SE and of receiving gene-positive risk information on intentions to quit. There was a significant Risk x SE interaction on intentions to attend an information session. SE was not associated with intentions to attend the information session for smokers in the gene-positive group. Intentions to attend the session were negatively associated with SE for smokers in the lower risk groups. Implications for using genetic risk information to motivate smoking cessation are discussed.

AB - Protection motivation theory and the extended parallel processing model are used to predict the motivational impact of information regarding a genetic susceptibility to heart disease. One hundred ninety-eight smokers read 1 of 3 vignettes: gene positive, gene negative, or standard smoking risk information. Analyses examined whether the impact of type of risk information was moderated by smokers' self-efficacy (SE) levels. Key outcomes were intention to quit and intention to attend an information session about quitting. There were significant main effects of SE and of receiving gene-positive risk information on intentions to quit. There was a significant Risk x SE interaction on intentions to attend an information session. SE was not associated with intentions to attend the information session for smokers in the gene-positive group. Intentions to attend the session were negatively associated with SE for smokers in the lower risk groups. Implications for using genetic risk information to motivate smoking cessation are discussed.

U2 - 10.1037/0278-6133.25.6.740

DO - 10.1037/0278-6133.25.6.740

M3 - Article

C2 - 17100502

VL - 25

SP - 740

EP - 752

JO - Health Psychology

JF - Health Psychology

SN - 0278-6133

ER -