Bilateral parietal dysfunctions and disconnections in simultanagnosia and Bálint syndrome

Research output: Chapter in Book/Report/Conference proceedingChapter (peer-reviewed)peer-review

Standard

Bilateral parietal dysfunctions and disconnections in simultanagnosia and Bálint syndrome. / Chechlacz, Magdalena.

The Parietal Lobe. Elsevier, 2018. p. 249-267 (Handbook of Clinical Neurology; Vol. 151).

Research output: Chapter in Book/Report/Conference proceedingChapter (peer-reviewed)peer-review

Harvard

APA

Vancouver

Author

Chechlacz, Magdalena. / Bilateral parietal dysfunctions and disconnections in simultanagnosia and Bálint syndrome. The Parietal Lobe. Elsevier, 2018. pp. 249-267 (Handbook of Clinical Neurology).

Bibtex

@inbook{7f980361f4fd402393f96a65fd6e99e8,
title = "Bilateral parietal dysfunctions and disconnections in simultanagnosia and B{\'a}lint syndrome",
abstract = "In 1909 Rezs{\"o} B{\'a}lint published an extraordinary case study of a man with complex visuospatial deficits resulting from bilateral parietal lesions. Despite some controversies over the nature of reported symptoms, in 1954 Hecaen and Ajuriaguerra conceived the term {"}B{\'a}lint syndrome,{"} not only to honor B{\'a}lint's influential work but to firmly conceptualize this striking neurologic disorder. Nowadays it is largely agreed that, while B{\'a}lint syndrome may result from multiple etiologies, it is principally diagnosed based on the presence of three symptoms: simultanagnosia, optic ataxia, and ocular apraxia. One of the most striking characteristics of B{\'a}lint syndrome, perfectly capturing the nature of this remarkable disorder, is that affected patients cannot perceive more than one object at a time or comprehend multiobject visual scenes due to a lack of ability to detect several objects concurrently and to grasp the spatial relationships between them. This chapter gives an overview of the cognitive mechanisms and neuroanatomy underlying B{\'a}lint syndrome, which provides key insights into our understanding of the role of parietal cortex in human attention, visual perception, and visuomotor control. This chapter also pays homage to Glyn Humphreys (1954-2016), who pioneered contributions to the knowledge about complexity of visual and spatial deficits associated with B{\'a}lint syndrome.",
author = "Magdalena Chechlacz",
note = "Copyright {\textcopyright} 2018 Elsevier B.V. All rights reserved.",
year = "2018",
month = mar,
day = "6",
doi = "10.1016/B978-0-444-63622-5.00012-7",
language = "English",
isbn = "978-0-444-63622-5",
series = "Handbook of Clinical Neurology",
publisher = "Elsevier",
pages = "249--267",
booktitle = "The Parietal Lobe",

}

RIS

TY - CHAP

T1 - Bilateral parietal dysfunctions and disconnections in simultanagnosia and Bálint syndrome

AU - Chechlacz, Magdalena

N1 - Copyright © 2018 Elsevier B.V. All rights reserved.

PY - 2018/3/6

Y1 - 2018/3/6

N2 - In 1909 Rezsö Bálint published an extraordinary case study of a man with complex visuospatial deficits resulting from bilateral parietal lesions. Despite some controversies over the nature of reported symptoms, in 1954 Hecaen and Ajuriaguerra conceived the term "Bálint syndrome," not only to honor Bálint's influential work but to firmly conceptualize this striking neurologic disorder. Nowadays it is largely agreed that, while Bálint syndrome may result from multiple etiologies, it is principally diagnosed based on the presence of three symptoms: simultanagnosia, optic ataxia, and ocular apraxia. One of the most striking characteristics of Bálint syndrome, perfectly capturing the nature of this remarkable disorder, is that affected patients cannot perceive more than one object at a time or comprehend multiobject visual scenes due to a lack of ability to detect several objects concurrently and to grasp the spatial relationships between them. This chapter gives an overview of the cognitive mechanisms and neuroanatomy underlying Bálint syndrome, which provides key insights into our understanding of the role of parietal cortex in human attention, visual perception, and visuomotor control. This chapter also pays homage to Glyn Humphreys (1954-2016), who pioneered contributions to the knowledge about complexity of visual and spatial deficits associated with Bálint syndrome.

AB - In 1909 Rezsö Bálint published an extraordinary case study of a man with complex visuospatial deficits resulting from bilateral parietal lesions. Despite some controversies over the nature of reported symptoms, in 1954 Hecaen and Ajuriaguerra conceived the term "Bálint syndrome," not only to honor Bálint's influential work but to firmly conceptualize this striking neurologic disorder. Nowadays it is largely agreed that, while Bálint syndrome may result from multiple etiologies, it is principally diagnosed based on the presence of three symptoms: simultanagnosia, optic ataxia, and ocular apraxia. One of the most striking characteristics of Bálint syndrome, perfectly capturing the nature of this remarkable disorder, is that affected patients cannot perceive more than one object at a time or comprehend multiobject visual scenes due to a lack of ability to detect several objects concurrently and to grasp the spatial relationships between them. This chapter gives an overview of the cognitive mechanisms and neuroanatomy underlying Bálint syndrome, which provides key insights into our understanding of the role of parietal cortex in human attention, visual perception, and visuomotor control. This chapter also pays homage to Glyn Humphreys (1954-2016), who pioneered contributions to the knowledge about complexity of visual and spatial deficits associated with Bálint syndrome.

U2 - 10.1016/B978-0-444-63622-5.00012-7

DO - 10.1016/B978-0-444-63622-5.00012-7

M3 - Chapter (peer-reviewed)

C2 - 29519461

SN - 978-0-444-63622-5

T3 - Handbook of Clinical Neurology

SP - 249

EP - 267

BT - The Parietal Lobe

PB - Elsevier

ER -