Absence of exercise-induced leptin suppression associated with insufficient epinephrine reserve in patients with classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency

Research output: Contribution to journalArticlepeer-review

Standard

Absence of exercise-induced leptin suppression associated with insufficient epinephrine reserve in patients with classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency. / Riepe, F G; Krone, N; Krüger, S N; Sweep, F C G J; Lenders, J W M; Dötsch, J; Mönig, H; Sippell, W G; Partsch, C-J.

In: Experimental and Clinical Endocrinology and Diabetes, Vol. 114, No. 3, 2006, p. 105-10.

Research output: Contribution to journalArticlepeer-review

Harvard

APA

Vancouver

Author

Bibtex

@article{7a4b5f3b723847ec8ea910003f1b7785,
title = "Absence of exercise-induced leptin suppression associated with insufficient epinephrine reserve in patients with classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency",
abstract = "Patients with congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency suffer from glucocorticoid and mineralocorticoid deficiency. They have insufficient epinephrine reserves and increased basal leptin levels and are often insulin resistant. In healthy subjects, an inhibitory effect of acute catecholamine elevation on the leptin plasma concentrations has been reported. However, it is not yet known how leptin levels respond to exercise in CAH patients.",
author = "Riepe, {F G} and N Krone and Kr{\"u}ger, {S N} and Sweep, {F C G J} and Lenders, {J W M} and J D{\"o}tsch and H M{\"o}nig and Sippell, {W G} and C-J Partsch",
year = "2006",
doi = "10.1055/s-2005-865836",
language = "English",
volume = "114",
pages = "105--10",
journal = "Experimental and Clinical Endocrinology and Diabetes",
issn = "0947-7349",
publisher = "Thieme Publishing",
number = "3",

}

RIS

TY - JOUR

T1 - Absence of exercise-induced leptin suppression associated with insufficient epinephrine reserve in patients with classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency

AU - Riepe, F G

AU - Krone, N

AU - Krüger, S N

AU - Sweep, F C G J

AU - Lenders, J W M

AU - Dötsch, J

AU - Mönig, H

AU - Sippell, W G

AU - Partsch, C-J

PY - 2006

Y1 - 2006

N2 - Patients with congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency suffer from glucocorticoid and mineralocorticoid deficiency. They have insufficient epinephrine reserves and increased basal leptin levels and are often insulin resistant. In healthy subjects, an inhibitory effect of acute catecholamine elevation on the leptin plasma concentrations has been reported. However, it is not yet known how leptin levels respond to exercise in CAH patients.

AB - Patients with congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency suffer from glucocorticoid and mineralocorticoid deficiency. They have insufficient epinephrine reserves and increased basal leptin levels and are often insulin resistant. In healthy subjects, an inhibitory effect of acute catecholamine elevation on the leptin plasma concentrations has been reported. However, it is not yet known how leptin levels respond to exercise in CAH patients.

U2 - 10.1055/s-2005-865836

DO - 10.1055/s-2005-865836

M3 - Article

C2 - 16636975

VL - 114

SP - 105

EP - 110

JO - Experimental and Clinical Endocrinology and Diabetes

JF - Experimental and Clinical Endocrinology and Diabetes

SN - 0947-7349

IS - 3

ER -