Abstract
The Type D personality has been associated with a range of negative health
outcomes including cardiovascular disease. A potential mechanism is large
magnitude cardiovascular reactivity to stress. However, the studies on reactivity to acute psychological stress in Type D individuals have reported equivocal findings, potentially due to the varying social aspects of the stressor employed. The present study, examined whether cardiovascular reactivity in Type D and Non-Type D undergraduate students differed according to an asocial (31 Type D , 30 Non-Type D: 52% female) or social (35 Type D, 34 Non-Type D: 55% female) version of the stress task. Type D personality was assessed using the DS14 questionnaire, with participant’s blood pressure and heart rate recorded at rest and during a 15-minute stress protocol comprising the Stroop and a mental arithmetic task. With adjustment for age, cardiovascular fitness, perceived stressfulness (which differed between the groups) and baseline levels (which did not differ between groups), there were significant group x condition interactions for systolic (p = .010) and diastolic (p = .029) blood pressure, and heart rate (p = .033) reactivity. Under the social condition, Type D individuals exhibited significantly greater systolic blood pressure (p = .010), and heart rate (p = .009) reactivity, with no group differences under the asocial condition. Diastolic blood pressure reactivity did not significantly vary according to Type D status within either condition. Interestingly, Type D individuals’ responses were somewhat lower than non-Type D individuals for all reactivity measures in the asocial condition, although this was not significant. This study highlights that Type D individuals only exhibit exaggerated haemodynamic reactions under conditions of high social evaluative threat. This suggests that the possible mechanism underlying the association between Type D personality and increased cardiovascular disease risk is via stress responses in highly social situations.
outcomes including cardiovascular disease. A potential mechanism is large
magnitude cardiovascular reactivity to stress. However, the studies on reactivity to acute psychological stress in Type D individuals have reported equivocal findings, potentially due to the varying social aspects of the stressor employed. The present study, examined whether cardiovascular reactivity in Type D and Non-Type D undergraduate students differed according to an asocial (31 Type D , 30 Non-Type D: 52% female) or social (35 Type D, 34 Non-Type D: 55% female) version of the stress task. Type D personality was assessed using the DS14 questionnaire, with participant’s blood pressure and heart rate recorded at rest and during a 15-minute stress protocol comprising the Stroop and a mental arithmetic task. With adjustment for age, cardiovascular fitness, perceived stressfulness (which differed between the groups) and baseline levels (which did not differ between groups), there were significant group x condition interactions for systolic (p = .010) and diastolic (p = .029) blood pressure, and heart rate (p = .033) reactivity. Under the social condition, Type D individuals exhibited significantly greater systolic blood pressure (p = .010), and heart rate (p = .009) reactivity, with no group differences under the asocial condition. Diastolic blood pressure reactivity did not significantly vary according to Type D status within either condition. Interestingly, Type D individuals’ responses were somewhat lower than non-Type D individuals for all reactivity measures in the asocial condition, although this was not significant. This study highlights that Type D individuals only exhibit exaggerated haemodynamic reactions under conditions of high social evaluative threat. This suggests that the possible mechanism underlying the association between Type D personality and increased cardiovascular disease risk is via stress responses in highly social situations.
Original language | English |
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Title of host publication | 72nd Annual Scientific Meeting |
Subtitle of host publication | Stretching the Boundaries: From Mechanisms of Disease to Models of Health |
Publisher | American Psychosomatic Society |
Pages | 17 |
Number of pages | 1 |
Publication status | Published - 13 Mar 2014 |
Event | American Psychosomatic Society - San Francisco, United States Duration: 12 Mar 2014 → 15 Mar 2014 |
Conference
Conference | American Psychosomatic Society |
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Country/Territory | United States |
City | San Francisco |
Period | 12/03/14 → 15/03/14 |