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Solving the RIDDLE of 53BP1 recruitment to sites of damage.
Grant Stewart
Cancer and Genomic Sciences
Research output
:
Contribution to journal
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Article
67
Citations (Scopus)
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Dive into the research topics of 'Solving the RIDDLE of 53BP1 recruitment to sites of damage.'. Together they form a unique fingerprint.
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Keyphrases
Double-strand Break Repair
100%
Repair Proteins
100%
DNA Double-strand Break Repair
100%
E3 Ubiquitin Ligase
100%
53BP1
100%
RNF168
100%
RNF8
100%
Cell-derived
50%
DNA Damage
50%
Cellular Response
50%
Phosphoinositide 3-kinase (PI3K)
50%
Immune System Development
50%
Ubiquitin
50%
Immunodeficiency
50%
DNA Double-strand Breaks
50%
Genetic Causes
50%
Neurodegeneration
50%
Delocalization
50%
Ubiquitylation
50%
Breast Cancer Susceptibility Gene 1 (BRCA1)
50%
Inherited mutation
50%
Genomic Instability
50%
DNA-dependent Protein Kinase (DNA-PK)
50%
Tumor Predisposition
50%
Premature Aging
50%
RIDDLE Syndrome
50%
Repair System
50%
DNA Repair-deficiency Disorders
50%
Ubiquitin System
50%
Integrated Network
50%
E3 Ligase
50%
Biochemistry, Genetics and Molecular Biology
Double-Strand DNA Break
100%
Ubiquitin
66%
Ubiquitin-Conjugating Enzyme
66%
RNF8
66%
Genome Instability
33%
Genetics
33%
DNA Damage
33%
Phosphoinositide 3-Kinase
33%
Ubiquitination
33%
DNA Repair
33%
BRCA1
33%
Ubiquitin Ligase
33%
Inherited Mutation
33%
Pharmacology, Toxicology and Pharmaceutical Science
Double Stranded DNA
100%
Ubiquitin
66%
Ubiquitin Protein Ligase E3
66%
Neoplasm
33%
Phosphatidylinositol 3 Kinase
33%
Syndrome
33%
Nerve Degeneration
33%
Disease Predisposition
33%
Immune Deficiency
33%
Ligase
33%
Premature Aging
33%
Disorders of DNA Synthesis and Repair
33%
Inherited Mutation
33%
Immunology and Microbiology
Ubiquitins
100%
Double-Strand DNA Break
75%
Immune System Development
25%
Immunodeficiency
25%
DNA Damage
25%
DNA Repair
25%
Genomic Instability
25%
Ubiquitination
25%