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Role of complement in myasthenia gravis
Pyae Phyo San
,
Saiju Jacob
*
*
Corresponding author for this work
Immunology and Immunotherapy
Research output
:
Contribution to journal
›
Review article
›
peer-review
106
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Keyphrases
Myasthenia Gravis
100%
Muscle Weakness
66%
Neuromuscular Junction
66%
Complement Therapy
66%
Randomized Controlled Trial
33%
IgG1
33%
IgG3
33%
Autoantibodies
33%
Clinical Course
33%
Receptor Complex
33%
Complement System
33%
Acetylcholine Receptor (AChR)
33%
Acetylcholine Receptor Antibody
33%
Prototypic
33%
Extraocular muscles
33%
Neuroimmune Diseases
33%
Paroxysmal Nocturnal Hemoglobinuria
33%
Bulbar muscles
33%
World Data
33%
Medicine and Dentistry
Myasthenia gravis
100%
Muscle Weakness
66%
Neuromuscular Junction
66%
Autoantibodies
33%
Randomized Controlled Trial
33%
Immunoglobulin G3
33%
Immunoglobulin G1
33%
Disease Course
33%
Weakness
33%
Complement System
33%
Cholinergic Receptor
33%
Cholinergic Receptor Antibody
33%
Diseases
33%
Real-World Data
33%
Paroxysmal Nocturnal Hemoglobinuria
33%
Pharmacology, Toxicology and Pharmaceutical Science
Myasthenia gravis
100%
Muscle Weakness
66%
Autoantibodies
33%
Randomized Controlled Trial
33%
Immunoglobulin G3
33%
Immunoglobulin G1
33%
Cholinergic Receptor
33%
Disease Course
33%
Weakness
33%
Cholinergic Receptor Antibody
33%
Paroxysmal Nocturnal Hemoglobinuria
33%
Diseases
33%
Neuroscience
Myasthenia gravis
100%
Neuromuscular Junction
66%
Cholinergic Receptor
33%
Autoantibodies
33%
Immunoglobulin G1
33%
Complement System
33%
Cholinergic Receptor Antibody
33%
Immunoglobulin G3
33%