Pathogenesis of non-alcoholic fatty liver disease.

Joanna Dowman; Jeremy Tomlinson; Philip Newsome

doi:10.1093/qjmed/hcp158

Pathogenesis of non-alcoholic fatty liver disease.

Joanna Dowman
, Jeremy Tomlinson
, Philip Newsome

Immunology and Immunotherapy

Research output: Contribution to journal › Article › peer-review

415 Citations (Scopus)

410 Downloads (Pure)

Abstract

Non-alcoholic fatty liver disease (NAFLD) represents a spectrum of disease ranging from hepatocellular steatosis through steatohepatitis to fibrosis and irreversible cirrhosis. The prevalence of NAFLD has risen rapidly in parallel with the dramatic rise in obesity and diabetes, and is rapidly becoming the most common cause of liver disease in Western countries. Indeed, NAFLD is now recognized to be the aetiology in many cases previously labelled as cryptogenic cirrhosis.

Original language	English
Pages (from-to)	71-83
Journal	QJM
Volume	103
Issue number	2
Early online date	13 Nov 2009
DOIs	https://doi.org/10.1093/qjmed/hcp158
Publication status	Published - 1 Feb 2010

Bibliographical note

null

Readers are entitled to use, reproduce, disseminate, or display the open access version of this article for non-commercial purposes provided that: the original authorship is properly and fully attributed; the journal and Oxford University Press are attributed as the original place of publication with the correct citation details given.

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

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10.1093/qjmed/hcp158Licence: Creative Commons: Attribution (CC BY)

Dowman.pdf

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2810391/

Cite this

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Pathogenesis of non-alcoholic fatty liver disease.

Abstract

Bibliographical note

UN SDGs

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