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Pathogenesis of ANCA-associated Vasculitis
Julia Flint, Matthew Morgan, Caroline Savage
Immunology and Immunotherapy
Research output
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Contribution to journal
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Article
26
Citations (Scopus)
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Dive into the research topics of 'Pathogenesis of ANCA-associated Vasculitis'. Together they form a unique fingerprint.
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Keyphrases
Neutrophils
100%
Antineutrophil Cytoplasmic Antibody
100%
Antineutrophil Cytoplasmic Antibody-associated Vasculitis
100%
Tissue Damage
100%
B Cells
50%
Signaling Pathway
50%
Cell-to-cell
50%
Cytokine Production
50%
Proteinase 3
50%
Neutrophil Activation
50%
Myeloperoxidase
50%
Degranulation
50%
Autoantibodies
50%
Regulatory Mechanism
50%
Endothelial Damage
50%
Target Antigen
50%
Pathogenic Mechanism
50%
Systemic Inflammatory Response
50%
Leukocyte-endothelial Interaction
50%
Inflammatory Vasculitis
50%
T Helper Cells
50%
Autoantibody Production
50%
Autoreactive
50%
Circulating Autoantibodies
50%
Neutrophil Granules
50%
Medicine and Dentistry
Autoantibodies
100%
Vasculitis
100%
Neutrophil
66%
Tissue Injury
66%
B Cell
33%
Signal Transduction
33%
Cytokine
33%
Myeloblastin
33%
Leukocyte Activation
33%
Myeloperoxidase
33%
Degranulation
33%
T-Helper Cell
33%
Cytokine Production
33%
Granulocyte Antibody
33%
Neutrophil Granule
33%
Immunology and Microbiology
Immunology
100%
Vasculitis
100%
Autoantibodies
75%
Signal Transduction
25%
B Cell
25%
Cytokine
25%
Cytokine Production
25%
Neutrophil Granulocyte
25%
Leukocyte Activation
25%
Neutrophil
25%
Degranulation
25%
T Helper Cell
25%
Neutrophil Granule
25%
Granulocyte Antibody
25%
Pharmacology, Toxicology and Pharmaceutical Science
Autoantibodies
100%
Vasculitis
100%
Cytokine
66%
Tissue Injury
66%
Myeloblastin
33%
Myeloperoxidase
33%
Granulocyte Antibody
33%