PAPSS2 deficiency causes androgen excess via impaired DHEA sulfation—in vitro and in vivo studies in a family harboring two novel PAPSS2 mutations

Wilma Oostdijk, Jan Idkowiak, Jonathan W. Mueller, Philip J. House, Angela E. Taylor, Michael W. O'reilly, Beverly A. Hughes, Martine C. De Vries, Sarina G. Kant, Gijs W. E. Santen, Annemieke J. M. H. Verkerk, André G. Uitterlinden, Jan M. Wit, Monique Losekoot, Wiebke Arlt

Research output: Contribution to journal › Article › peer-review

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The Role of DHEA Sulfation in the Pre-Receptor Regulation of Androgen Excess
Arlt, W.
THE WELLCOME TRUST
1/10/11 → 30/09/14
Project: Research
The role of DHEA sulfation in childhood androgen excess
Idkowiak, J., Arlt, W. & Barrett, T.
Medical Research Council
1/05/11 → 30/04/13
Project: Research Councils
Pre-Receptor Regulation of Dehydroepiandrosterone Synthesis, Metabolism and Action
Arlt, W.
Medical Research Council
1/08/04 → 31/10/09
Project: Research Councils

PAPSS2 deficiency causes androgen excess via impaired DHEA sulfation—in vitro and in vivo studies in a family harboring two novel PAPSS2 mutations

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The Role of DHEA Sulfation in the Pre-Receptor Regulation of Androgen Excess

The role of DHEA sulfation in childhood androgen excess

Pre-Receptor Regulation of Dehydroepiandrosterone Synthesis, Metabolism and Action