Skip to main navigation
Skip to search
Skip to main content
University of Birmingham Home
Help & FAQ
Link opens in a new tab
Search content at University of Birmingham
Home
Research output
Profiles
Research units
Projects
Activities
Datasets
Equipment
Prizes
Press/Media
hMSH2 splice site mutation and endometrial cancer
F Bianchi
, S Rosati
, L Belvederesi
, C Loretelli
, R Catalani
, A Mandolesi
, R Bracci
, I Bearzi
, Emilio Porfiri
, R Cellerino
Cancer Research UK Clinical Trials Unit
Research output
:
Contribution to journal
›
Article
7
Citations (Scopus)
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'hMSH2 splice site mutation and endometrial cancer'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Splicing mutation
100%
Endometrial Cancer
100%
Hereditary Colorectal Cancer Syndromes
100%
HMSH2
100%
MSH2 Gene
75%
Gene mutation
50%
Germ Cells
50%
Mismatch Repair Genes
50%
MSH2
50%
Cancer Susceptibility
50%
Endometrial Tumor
50%
High Risk
25%
Tumor
25%
Colon Cancer
25%
Early Onset
25%
Protein Expression
25%
Colorectal Cancer Patients
25%
Microsatellite Instability-high (MSI-H)
25%
Germline mutation
25%
Endometrium
25%
Tumor Development
25%
Colorectal Cancer Risk
25%
Inherited Syndromes
25%
Extracolonic Malignancy
25%
PTEN Inactivation
25%
Familial History
25%
Mismatch Repair
25%
Stop Signal
25%
Endometrial Cancer Screening
25%
Messenger RNA Processing
25%
Cancer Families
25%
Medicine and Dentistry
Uterine Cancer
100%
Hereditary Nonpolyposis Colorectal Cancer
100%
DNA Mismatch Repair
75%
Gene Mutation
50%
Germ Cell
50%
Cancer Susceptibility
50%
Germ Line
50%
Endometrium Tumor
50%
DNA Mismatch Repair Protein MSH2
50%
Malignant Neoplasm
25%
Neoplasm
25%
Colorectal Carcinoma
25%
Colon Carcinoma
25%
Protein Expression
25%
Messenger RNA
25%
Cancer
25%
Prematurity
25%
Microsatellite Instability
25%
Germline Mutation
25%
Endometrium
25%
Endometrial Cancer Screening
25%
RNA Processing
25%