Genomic characterisation of the chromosomal breakpoints of the t(4;14) of multiple myeloma suggest more than one possible aetiological mechanism

JAL Fenton; Guy Pratt; AC Rawstron; K Sibley; D Rothwell; Z Yates; A Dring; S Richards; JA Ashcroft; FE Davies; RJ Owen; JA Child; GJ Morgan

doi:10.1038/sj.onc.1206335

Genomic characterisation of the chromosomal breakpoints of the t(4;14) of multiple myeloma suggest more than one possible aetiological mechanism

JAL Fenton, Guy Pratt, AC Rawstron, K Sibley, D Rothwell, Z Yates, A Dring, S Richards, JA Ashcroft, FE Davies, RJ Owen, JA Child, GJ Morgan

Cancer and Genomic Sciences

Research output: Contribution to journal › Article

20 Citations (Scopus)

Original language	English
Pages (from-to)	1103-1113
Number of pages	11
Journal	Oncogene
Volume	22
Issue number	7
DOIs	https://doi.org/10.1038/sj.onc.1206335
Publication status	Published - 20 Feb 2003

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10.1038/sj.onc.1206335

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Fenton, JAL., Pratt, G., Rawstron, AC., Sibley, K., Rothwell, D., Yates, Z., Dring, A., Richards, S., Ashcroft, JA., Davies, FE., Owen, RJ., Child, JA., & Morgan, GJ. (2003). Genomic characterisation of the chromosomal breakpoints of the t(4;14) of multiple myeloma suggest more than one possible aetiological mechanism. Oncogene, 22(7), 1103-1113. https://doi.org/10.1038/sj.onc.1206335

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Fenton, JAL, Pratt, G, Rawstron, AC, Sibley, K, Rothwell, D, Yates, Z, Dring, A, Richards, S, Ashcroft, JA, Davies, FE, Owen, RJ, Child, JA & Morgan, GJ 2003, 'Genomic characterisation of the chromosomal breakpoints of the t(4;14) of multiple myeloma suggest more than one possible aetiological mechanism', Oncogene, vol. 22, no. 7, pp. 1103-1113. https://doi.org/10.1038/sj.onc.1206335

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AU - Fenton, JAL

AU - Pratt, Guy

AU - Rawstron, AC

AU - Sibley, K

AU - Rothwell, D

AU - Yates, Z

AU - Dring, A

AU - Richards, S

AU - Ashcroft, JA

AU - Davies, FE

AU - Owen, RJ

AU - Child, JA

AU - Morgan, GJ

PY - 2003/2/20

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