Cold acclimation induces physiological cardiac hypertrophy and increases assimilation of triacylglycerol metabolism through lipoprotein lipase.

Yuzhu Cheng; David Hauton

doi:10.1016/j.bbalip.2008.07.006

Cold acclimation induces physiological cardiac hypertrophy and increases assimilation of triacylglycerol metabolism through lipoprotein lipase.

Yuzhu Cheng, David Hauton

Cardiovascular Sciences

Research output: Contribution to journal › Article

16 Citations (Scopus)

Abstract

The contribution of triacylglycerol to energy provision in the hypertrophied heart, mediated through lipoprotein lipase (LPL) is largely unknown and the contribution of very-low-density lipoprotein (VLDL) receptor to control of LPL presentation at the endothelium is unclear. For isolated perfused rat hearts, cold acclimation (CA) induced volume-overload hypertrophy, with decreased developed pressure (P

Original language	English
Pages (from-to)	618-26
Number of pages	9
Journal	Biochimica et Biophysica Acta
Volume	1781
Issue number	10
DOIs	https://doi.org/10.1016/j.bbalip.2008.07.006
Publication status	Published - 1 Oct 2008

Keywords

Cardiac hypertrophy
AMPK
Very-low-density lipoprotein receptor
Triacylglycerol

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10.1016/j.bbalip.2008.07.006

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abstract = "The contribution of triacylglycerol to energy provision in the hypertrophied heart, mediated through lipoprotein lipase (LPL) is largely unknown and the contribution of very-low-density lipoprotein (VLDL) receptor to control of LPL presentation at the endothelium is unclear. For isolated perfused rat hearts, cold acclimation (CA) induced volume-overload hypertrophy, with decreased developed pressure (P",

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AU - Cheng, Yuzhu

AU - Hauton, David

PY - 2008/10/1

Y1 - 2008/10/1

N2 - The contribution of triacylglycerol to energy provision in the hypertrophied heart, mediated through lipoprotein lipase (LPL) is largely unknown and the contribution of very-low-density lipoprotein (VLDL) receptor to control of LPL presentation at the endothelium is unclear. For isolated perfused rat hearts, cold acclimation (CA) induced volume-overload hypertrophy, with decreased developed pressure (P

AB - The contribution of triacylglycerol to energy provision in the hypertrophied heart, mediated through lipoprotein lipase (LPL) is largely unknown and the contribution of very-low-density lipoprotein (VLDL) receptor to control of LPL presentation at the endothelium is unclear. For isolated perfused rat hearts, cold acclimation (CA) induced volume-overload hypertrophy, with decreased developed pressure (P

KW - Cardiac hypertrophy

KW - AMPK

KW - Very-low-density lipoprotein receptor

KW - Triacylglycerol

UR - https://www.scopus.com/pages/publications/52049127012

U2 - 10.1016/j.bbalip.2008.07.006

DO - 10.1016/j.bbalip.2008.07.006

M3 - Article

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VL - 1781

SP - 618

EP - 626

JO - Biochimica et Biophysica Acta

JF - Biochimica et Biophysica Acta

IS - 10

ER -

Cold acclimation induces physiological cardiac hypertrophy and increases assimilation of triacylglycerol metabolism through lipoprotein lipase.

Abstract

Keywords

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