Abnormal angiopoetins 1 & 2, angiopoetin receptor Tie-2 and vascular endothelial growth factor levels in hypertension: relationship to target organ damage (a sub-study of the Anglo-Scandinavian Cardiac Outcomes Trial ASCOT)

Sunil Nadar; Andrew Blann; David Beevers; Gregory Lip

doi:10.1111/j.1365-2796.2005.01550.x

Abnormal angiopoetins 1 & 2, angiopoetin receptor Tie-2 and vascular endothelial growth factor levels in hypertension: relationship to target organ damage (a sub-study of the Anglo-Scandinavian Cardiac Outcomes Trial ASCOT)

Sunil Nadar, Andrew Blann, David Beevers, Gregory Lip

Research output: Contribution to journal › Article

75 Citations (Scopus)

Original language	English
Pages (from-to)	336-343
Number of pages	8
Journal	Journal of Internal Medicine
Volume	258
DOIs	https://doi.org/10.1111/j.1365-2796.2005.01550.x
Publication status	Published - 1 Oct 2005

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10.1111/j.1365-2796.2005.01550.x

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Nadar, S., Blann, A., Beevers, D., & Lip, G. (2005). Abnormal angiopoetins 1 & 2, angiopoetin receptor Tie-2 and vascular endothelial growth factor levels in hypertension: relationship to target organ damage (a sub-study of the Anglo-Scandinavian Cardiac Outcomes Trial ASCOT). Journal of Internal Medicine, 258, 336-343. https://doi.org/10.1111/j.1365-2796.2005.01550.x

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Nadar, S, Blann, A, Beevers, D & Lip, G 2005, 'Abnormal angiopoetins 1 & 2, angiopoetin receptor Tie-2 and vascular endothelial growth factor levels in hypertension: relationship to target organ damage (a sub-study of the Anglo-Scandinavian Cardiac Outcomes Trial ASCOT)', Journal of Internal Medicine, vol. 258, pp. 336-343. https://doi.org/10.1111/j.1365-2796.2005.01550.x

Abnormal angiopoetins 1 & 2, angiopoetin receptor Tie-2 and vascular endothelial growth factor levels in hypertension: relationship to target organ damage (a sub-study of the Anglo-Scandinavian Cardiac Outcomes Trial ASCOT). / Nadar, Sunil; Blann, Andrew; Beevers, David et al.
In: Journal of Internal Medicine, Vol. 258, 01.10.2005, p. 336-343.

Research output: Contribution to journal › Article

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